📊 Definition & Aetiology
Obstruction to flow into or out of the heart → decreased cardiac output. Types: extrinsic (tamponade, tension pneumothorax, PE) vs intrinsic (duct-dependent systemic lesions: HLHS, critical AS, coarctation).
🩸 Pathophysiology
Impaired diastolic filling (tamponade) or excessive afterload (PE, tension pneumothorax) → decreased CO → shock. Raised JVP/CVP is hallmark. Ventricular interdependence worsens LV filling.
🫀 Pericardial Tamponade
Beck's triad: hypotension, muffled heart sounds, raised JVP. Pulsus paradoxus (>10 mmHg inspiratory fall). ECHO: diastolic RA/RV collapse. Treatment: pericardiocentesis.
🫁 Tension Pneumothorax
Unilateral absent breath sounds, hyperresonance, tracheal deviation, hypotension. Treatment: needle decompression (2nd ICS MCL) → chest tube.
🩸 Massive Pulmonary Embolism
Sudden dyspnoea, hypoxia, hypotension, RV strain on ECHO. High-risk PE: thrombolysis or embolectomy.
❤️ Duct-Dependent Systemic Circulation
Neonates present with shock within days of birth as PDA closes (HLHS, critical AS, coarctation). Treatment: PGE1 infusion 0.05-0.1 mcg/kg/min to reopen PDA.