📋 MOCK OSCE · FCPS, MCPS, MD PAEDIATRICS ⏱ 10 min · CELIAC DISEASE

Celiac Disease · Short Case

Candidate task: perform focused clinical examination (GPE + abdomen) on a child with suspected celiac disease.
Then discuss differential diagnosis, investigations, management & follow‑up.
Pre‑exam Protocol
· Wash, Warm, Introduce, Position, Expose, Approach

Standard pre‑examination protocol – must be demonstrated:

🖐 Wash hands with sterilizing solution (or alcohol gel).
🔥 Warm hands and stethoscope before touching the child.
👋 Introduce yourself to the child and parent/carer.
🧍 Position the child appropriately: standing → sitting → lying supine.
👕 Exposure — remove shirt/top; roll up trousers to expose abdomen and lower limbs.
➡️ Approach from the right side (standard for abdominal examination).
CPSP marker: Pre‑exam Protocol is observed and scored. Always comment on what you are doing.
1. Clinical Examination (≈6 min)
02 General Physical Exam & Anthropometry

Key areas: growth, nutrition, and extra‑intestinal signs.

  • Anthropometry: weight, height/length, BMI, head circumference (if <2y). Plot on WHO/CDC charts — look for falling weight/height percentiles (classic “growth faltering”).
  • Nutritional status: muscle wasting, reduced subcutaneous fat, loose skinfolds, abdominal distension (pot‑belly).
  • Skin: pallor (anaemia), dermatitis herpetiformis (vesicular, intensely pruritic on extensor surfaces), bruising (vitamin K deficiency).
  • Oral: aphthous stomatitis, dental enamel hypoplasia (pitting/discoloration).
  • MSK: bone pain, rickets (vitamin D malabsorption), delayed puberty.
  • Neurological: peripheral neuropathy, ataxia (vitamin E deficiency – rare but serious).
📏 Anthropometry clue: weight affected first, then height; head circumference usually spared unless severe/prolonged.
03 Abdominal Examination

Systematic approach: inspection, auscultation, palpation, percussion.

  • Inspection: distension (often marked in children with celiac disease), visible peristalsis, muscle wasting, umbilicus (everted).
  • Auscultation: hyperactive bowel sounds (malabsorption) or normal; bruits rare.
  • Palpation: generally soft, non‑tender; may feel doughy; no masses (unless complicated).
  • Percussion: tympanic (gas from fermentation) but can be dull if ascites (rare).
  • Rectal: not routinely needed; may be deferred unless bleeding or constipation.
🔍 Key finding: abdominal distention with normal or hyperactive bowel sounds in a child with growth failure → strongly suggests malabsorption.
04 Summary of Clinical Findings

Expected findings in a child with untreated celiac disease:

SystemFindings
GrowthWeight & height below 3rd–5th percentile or crossing major percentiles downward; BMI low.
NutritionMuscle wasting (gluteal, quadriceps), loss of subcutaneous fat, loose skinfolds, abdominal distension.
SkinPallor (anaemia), dermatitis herpetiformis (if present – highly specific), ecchymoses (vitamin K).
OralAphthous ulcers, enamel hypoplasia (permanent teeth).
AbdomenDistended, tympanitic, hyperactive bowel sounds; non‑tender; no organomegaly (unless associated liver disease).
NeurologicalAtaxia, peripheral neuropathy (vitamin E deficiency – late).
MSKBone pain, rickets (vitamin D deficiency), delayed puberty.
🧠 Interpretation: growth failure + abdominal distension + pallor + (possibly) dermatitis herpetiformis → classic celiac disease presentation. However, many present with non‑classical features (anaemia, short stature, fatigue).

📋 Case Presentation

A 3‑year‑old girl presents with abdominal distension, clubbing, and poor weight gain On examination, the child is irritable, has muscle wasting and a protuberant abdomen. There is no fever or significant abdominal tenderness. Growth parameters show weight <3rd percentile and height at the 10th percentile.

2. Viva Discussion (≈4 min)
05 Viva · Differential, Investigations, Management, Follow‑up

Examiner will probe: differential diagnosis, diagnostic approach, treatment, and long‑term care.

🔹 Differential Diagnosis

• Cow’s milk protein allergy / enteropathy
Negative points: onset usually within first months of life, not after gluten introduction; usually resolves with milk elimination; no specific antibodies (tTG normal).

• Post‑infectious enteropathy (giardiasis, viral)
Negative points: acute onset, often self‑limiting; giardiasis may have foul‑smelling diarrhoea but no growth failure; tTG normal; stool microscopy positive.

• Cystic fibrosis (pancreatic insufficiency)
Negative points: associated with recurrent chest infections, meconium ileus, clubbing; sweat chloride >60 mEq/L; normal tTG.

• Crohn disease
Negative points: often has abdominal pain, bloody diarrhoea, perianal disease; elevated CRP/ESR; normal tTG; endoscopy shows skip lesions.

• Autoimmune enteropathy (rare)
Negative points: very early onset (<6 months); associated with other autoimmune diseases; anti‑enterocyte antibodies; normal tTG.

• Immunodeficiency (CVID, IgA deficiency)
Negative points: recurrent infections, low immunoglobulins; tTG may be falsely low in IgA deficiency (use IgG‑based testing).

• Tropical sprue / environmental enteropathy
Negative points: history of travel to endemic areas; responds to antibiotics; tTG normal.

• Carbohydrate malabsorption (lactose, sucrose‑isomaltase)
Negative points: isolated carbohydrate intolerance; no growth failure; stool pH <5.5 with reducing substances; tTG normal.

🔹 Key Investigations

Serology (first line):
• Anti‑tissue transglutaminase IgA (tTG‑IgA) – sensitivity >95%
• Total serum IgA (to rule out IgA deficiency)
• If IgA deficient: tTG‑IgG or deamidated gliadin peptide (DGP) IgG
• Endomysial antibody (EMA) – confirmatory if tTG borderline

Small bowel biopsy (gold standard):
• At least 4–6 biopsies from duodenum (including bulb)
• Marsh score ≥ 2 (increased IEL) or 3 (villous atrophy)

Supporting tests:
• CBC (anaemia – iron, folate, B12), ferritin, folate, vitamin B12
• Vitamin D, calcium, ALP (bone health), LFTs
• HLA‑DQ2/DQ8 (if diagnostic uncertainty, or on GFD)

🔹 Management Plan

Lifelong gluten‑free diet (GFD):
• Strict avoidance of wheat, rye, barley (and contaminated oats).
• Dietitian referral – essential for education and monitoring.
• Gluten‑free substitutes: rice, corn, quinoa, buckwheat, millet.

Nutritional rehabilitation:
• Correct micronutrient deficiencies: iron, folate, B12, vitamin D, calcium.
• Monitor weight and height gain; catch‑up growth expected.
• Consider lactose restriction initially (secondary lactase deficiency).

Follow‑up:
• Repeat tTG‑IgA at 6–12 months (should decrease).
• Clinical response (symptom resolution, growth improvement).
• If no response: reassess dietary compliance, consider refractory celiac disease.

🔹 Follow‑up & Complications

Short‑term (3–6 months):
• Symptom improvement (abdominal pain, diarrhoea, energy).
• Weight gain and growth velocity.
• Repeat tTG‑IgA (should fall by 50% or more).

Long‑term (annually):
• Monitor growth, BMI, and pubertal development.
• Check haematinics, vitamin D, calcium, LFTs, thyroid function (associated autoimmune conditions).
• Bone density (DEXA) if prolonged disease or poor adherence.

Complications if untreated:
• Growth failure, delayed puberty, osteoporosis.
• Other autoimmune diseases (type 1 diabetes, thyroiditis).
• Rare: intestinal lymphoma (enteropathy‑associated T‑cell lymphoma), refractory celiac disease.

📌 Additional viva topics:

  • When to perform HLA‑DQ2/DQ8 testing?
  • How to interpret tTG‑IgA levels (≥10x ULN → may avoid biopsy in children).
  • Role of endoscopy vs serology in diagnosis.
  • Management of non‑responsive celiac disease.
  • Screening of first‑degree relatives.
  • Associated conditions (type 1 DM, Down syndrome, IgA deficiency).
💡 Examiner expectation: logical linkage between clinical findings, serology, and biopsy; clear management plan with dietitian involvement; awareness of complications and need for lifelong follow‑up.
Mock OSCE · Celiac Disease · Based on Wyne‑Harris, Nelson & Pediatric Clinical Advisor